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BerGenBio

Regulatory Filings Apr 13, 2018

3555_rns_2018-04-13_3a5ee58a-c17d-4fd2-98da-6a2273f27e9f.html

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Promising pre-clinical data supporting BerGenBio's pipeline to be published and presented at upcoming leading conferences

Promising pre-clinical data supporting BerGenBio's pipeline to be published and presented at upcoming leading conferences

· Bemcentinib active in pre-clinical models of IPF and NASH

· IPF and cirrhotic NASH patients with aggressive disease identified by AXL

blood based biomarker

· BGB601 (partnered AXL ADC drug candidate) anti tumour pre-clinical data to

be presented at AACR.

Bergen, Norway, 13 April 2018 - BerGenBio ASA (OSE: BGBIO) announces that

promising pre-clinical data demonstrating that selective AXL inhibition

counteracts the progression of aggressive fibrosis in the lung and liver has

been published in the international peer-reviewed journal American Journal of

Respiratory and Critical Care Medicine (AJRCCM, (1)) and accepted for

presentation at the 53rd annual meeting of the European Association for the

Study of the Liver (EASL), respectively.

Additionally, pre-clinical data of BerGenBio's out-licensed AXL ADC candidate

BGB601 has been accepted for presentation at the American Association for Cancer

Research (AACR) Annual Meeting 2018.

The details of the publications are as follows:

Potential of selective AXL inhibition as a mechanism for treating the

progressive, rare, and frequently fatal lung disease idiopathic pulmonary

fibrosis (IPF):

Milena Espindola and colleagues from Cedars-Sinai in Los Angeles, CA, published

evidence in the American Journal of Respiratory and Critical Care Medicine

(AJRCCM) that AXL receptor expression is significantly elevated in patient

cells, tissues and models of IPF. Consistent with the pathological role of AXL

in fibrosis, selective inhibition of AXL using bemcentinib (BGB324) impacted IPF

fibroblast functions and the development of fibrosis in pre-clinical models of

IPF. The data also show a clear distinction in AXL levels between fast and slow

progressing IPF patients, highlighting the potential of using AXL levels as a

biomarker to (i) identify patients with a poor prognosis and who may respond to

treatment with an AXL inhibitor, and (ii) enrich patient populations in future

clinical trials.

The article in press is entitled: "Targeting of TAM Receptors Ameliorates

Fibrotic Mechanisms in Idiopathic Pulmonary Fibrosis" and can be accessed at the

AJRCCM's website - click

here (http://www.atsjournals.org/doi/abs/10.1164/rccm.201707-1519OC).

Potential of selective AXL inhibition for treating advanced form of non

-alcoholic steatohepatitis (NASH)

Anna Tutusaus et al. will be presenting a poster on AXL targeting in NASH

entitled "AXL increase in NASH patients and anti-fibrotic efficacy of AXL

inhibition in experimental NASH" (abstract: FRI-074) at the European Association

for the Study of the Liver (EASL) Annual Meeting in Paris, France, on Friday 13

April between 9 and 17:00 CET.

The abstract is available online - click here (https://program.m

-anage.com/ProgramSearch/?eventName=ilc2018&language=en

-GB&searchString=axl&pProgramGrade=Scientific).

Pre-clinical data in models of aggressive, AXL expressing, tumours supporting

clinical development of AXL ADC BGB601 (ADCT-601)

Lorenzo Zammarchi et al. will be presenting a poster on the in vitro and in vivo

anti-tumour activity of BGB601 (ADCT-601) in human cancer cell lines and animal

models as well as data on its safety and tolerability entitled "Preclinical

activity of ADCT-601, a novel pyrrolobenzodiazepine (PBD) dimer-based antibody

-drug conjugate (ADC) targeting AXL-expressing tumors" (abstract: 2792A) at the

American Association of Cancer Research (AACR) Annual Meeting in Chicago, IL, on

Monday 16 April between 13:00 and 17:00 CDT.

The abstract is available online at the AACR Annual Meeting website - click

here (http://www.abstractsonline.com/pp8/#!/4562/presentation/9909).

Richard Godfrey, CEO of BerGenBio commented: "This patient data and pre-clinical

findings in IPF and cirrhotic NASH are very compelling and suggest that

selective AXL inhibition may have potential as a new approach to treating life

-threatening fibrotic diseases. While our focus remains clearly on completing

our phase II clinical programme with bemcentinib and to establish proof of

concept for its role as a cornerstone of cancer therapy, we are intrigued by the

possibility of therapeutic benefit from our AXL inhibitors in fibrotic diseases.

We will continue to support this research and look forward to integrating it

into our pipeline development strategies. Furthermore, we are encouraged by pre

-clinical data supporting the advancement of BGB601, our out-licensed AXL ADC

drug candidate, towards the clinic."

-End-

About fibrosis, IPF and NASH

Fibrosis is an exaggerated healing response that fails to terminate

appropriately causing almost half of fatalities across all non-communicable

diseases combined.

Idiopathic Pulmonary Fibrosis (IPF) is a severe, progressive disease

characterised by fibrosis in the lung. Approximately 12 in 100,000 people will

develop IPF each year, the current approved treatments may slow down progression

of the disease but are not curative. Prognosis for those diagnosed with IPF is

poor, with median survival of only 2-3 years (2).

Non-alcoholic steatohepatitis (NASH) is a type of fatty liver disease

characterised by fibrosis, inflammation and liver cell damage for which there is

no approved treatment. Across the US and EU5, approximately 25 million people

are believed to have NASH - a proportion of whom will experience significant

worsening of their condition eventually leading to fatal liver failure.

About BGB601 (ADCT-601)

BGBC601 (ADCT-601) is an Antibody Drug Conjugate (ADC) drug, composed of a

humanised IgG1 antibody against human AXL, discovered at BerGenBio and out

-licensed for further development by ADC Therapeutics

SA (http://adctherapeutics.com).

About the EASL and AACR Annual Meetings

The EASL Annual Meeting is the largest congress dedicated to hepatology. 10,000

international delegates from a wide range of scientific disciplines gather to

present the latest research at this highly regarded event. The 2018 EASL Annual

meeting will take place in Paris, France, 11 - 15 April. For more information

please visit: https://ilc-congress.eu.

The AACR is the largest professional organisation in the world dedicated to

advancing cancer research and its mission to prevent and cure all cancers.

During the 2018 annual meeting, thousands of oncology researchers and clinicians

will gather in Chicago, IL, April 14 - 18. For more information, please visit:

http://www.aacr.org

About BerGenBio ASA

BerGenBio ASA is a clinical-stage biopharmaceutical company focused on

developing a pipeline of first-in-class AXL kinase inhibitors as a potential

cornerstone of combination cancer therapy. The Company is a world leader in

understanding the essential role of AXL kinase in mediating cancer spread,

immune evasion and drug resistance in multiple aggressive solid and

haematological cancers.

BerGenBio's lead product, bemcentinib (BGB324), is a selective, potent and

orally bio-available small molecule AXL inhibitor in four Company sponsored

Phase II clinical trials in major cancer indications, with read-outs anticipated

during 2018. It is the only selective AXL inhibitor in clinical development.

The Company sponsored clinical trials are:

· Bemcentinib with TARCEVA® (erlotinib) in advanced EGFR mutation driven non

-small cell lung cancer (NSCLC)

· Bemcentinib with KEYTRUDA in advanced adenocarcinoma of the lung, and

· Bemcentinib with KEYTRUDA in triple-negative breast cancer (TNBC).

· Bemcentinib as a single agent and combination therapy in acute myeloid

leukaemia (AML) / myeloid dysplastic syndrome (MDS)

The clinical trials combining bemcentinib with KEYTRUDA in adenocarcinoma of the

lung and TNBC are conducted in collaboration with Merck & Co., Inc. (Kenilworth,

NJ, USA), through a subsidiary.

In addition, a number of investigator-sponsored trials are underway, including a

trial to investigate bemcentinib with either MEKINIST® (trametinib) plus

TAFINLAR® (dabrafenib) or KEYTRUDA in advanced melanoma, as well as a trial

combining bemcentinib with docetaxel in advanced NSCLC.

BerGenBio is simultaneously developing a companion diagnostic test to identify

patient subpopulations most likely to benefit from treatment with bemcentinib.

This will facilitate more efficient registration trials and support a precision

medicine based commercialization strategy.

The Company is also developing a diversified pre-clinical pipeline of drug

candidates, including BGB149, an anti-AXL monoclonal antibody.

For further information, please visit: www.bergenbio.com

KEYTRUDA® is a registered trademark of Merck Sharp & Dohme Corp., a subsidiary

of Merck & Co., Inc., Kenilworth, NJ, USA, TARCEVA® is a registered trademark of

OSI Pharmaceuticals, LLC., marketed by Roche-Genentech. TAFLINAR® is a

registered trademark of Novartis International AG and MEKINIST® is a registered

trademark of GSK plc.

References

(1) Espindola MS et al AJRCC 2018

(2) Raghu G et al AJRCCM 2011

Contacts

Richard Godfrey

CEO, BerGenBio ASA

+47 917 86 304

Rune Skeie, CFO, BerGenBio ASA

[email protected]

+47 917 86 513

Media Relations in Norway

Jan Petter Stiff, Crux Advisors

[email protected]

+47 995 13 891

International Media Relations

David Dible, Mark Swallow, Marine Perrier, Citigate Dewe Rogerson

[email protected]

+44 207 638 9571

Forward looking statements

This announcement may contain forward-looking statements, which as such are not

historical facts, but are based upon various assumptions, many of which are

based, in turn, upon further assumptions. These assumptions are inherently

subject to significant known and unknown risks, uncertainties and other

important factors. Such risks, uncertainties, contingencies and other important

factors could cause actual events to differ materially from the expectations

expressed or implied in this announcement by such forward-looking statements

This information is subject to the disclosure requirements pursuant to section 5

-12 of the Norwegian Securities Trading Act.

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